an old saying that says, "If you want to find the best solution ,
find someone who is already getting the results you want". For the
past eleven years I have been constantly asked how I have accomplished
something few have been able to do. It seems that most people know of
somebody who has IBD, that is, Crohn's Disease or Ulcerative Colitis or they have it themselves. So getting well is what
this is about. Because of liability I cannot say that I can help you
"cure" anything. I am only recommending some measures you can
take that will eliminate your symptoms. If you need references of others
that have turned their life around I will be glad to oblige.
I will discuss what causes Inflammatory bowel disease and I will
discuss the protocol to follow based upon information gathered from the
scientific and medical communities. Then I will give some personal
insights and finally the resources where I got my information.
If you or somebody you know is suffering from any digestive ailments
I hope that after reading this you ask yourself, "Does this make
sense?" and then if it does, try it and see the results for yourself.
This is not meant as a replacement for your doctors care but I want
you to have this information available to you as an option. This
information is not my opinion. It is based upon a lot of research. If your
physician is not open to suggestions and is not getting results that you
think are possible than I suggest that you find a M.D. that will work with
you and has an open mind to other alternatives. I know the suffering that
people with IBD goes through and the goal here is to eliminate the causes
of these maladies. I do not understand somebody standing in the way if
there is even a remote chance that something might work.
The debilitating intestinal problems seen today have existed for
centuries. The names given the various conditions with the symptoms of
diarrhea, excess gas, loss of weight, excess mucus, cramping, blood loss,
and severe constipation have changed through the years. The method of
diagnosis as well as those of treatment and management have also changed
with time. But always, there has been a strong underlying belief that diet
is an important factor to consider, not only in determining the causes of
the disorders, but also in their treatment and cure. As far back as 1745,
Prince Charles, the Young Pretender to the throne of England, suffered
from ulcerative colitis and was said to have cured himself by adopting a
In the early 1900,s, physicians brought insight to our understanding
of the effect of food on intestinal problems. Dr. Christian Herter, a
physician and professor at Columbia University, noted that in every case
where children were wasting away with diarrhea and debilitation, proteins
were well tolerated, fats were handled moderately well but carbohydrates
(sugars and starches) were badly tolerated. He noted that eating some
carbohydrates almost invariably caused a relapse or a return of diarrhea
after a period of improvement. About that time Dr. Samuel Gee, another
world renowned children's specialist said that if the patient with
intestinal disease could be cured at all, it would have to be by means of
diet. Dr. Gee stated, " We must never forget that what the patient
takes beyond his power to digest does harm."
So what causes this inability to digest certain foods and how does
this lead to inflammatory conditions in the bowel? It is generally
accepted among physicians and researchers that during intestinal upsets
and chronic intestinal disease, the normal, harmonious state of balance
between intestinal microbes living in our gastrointestinal tract is lost.
The gastrointestinal tract becomes populated right after birth with
various types of microbes depending on the type of milk ingested as well
as other environmental factors. Studies have revealed that eventually more
than four hundred bacterial species live together in the human colon.
In the healthy intestinal tract, intestinal microbes appear to live
in a state of balance; an over abundance of one type seems to be inhibited
by the activities of other types. This competition between microbes
prevents any one type from overwhelming the body with its waste products
or toxins. An important protective factor which keeps the sparse bacterial
population of the stomach and upper small intestine is the high acidity of
the stomach's hydrochloric acid in which microbes cannot usually survive.
However, bacterial overgrowth can occur for various reasons, among
(1) Interference with the high acidity of the stomach through the
continual use of antacids.
(2) Malnutrition or a diet of poor quality, and the resulting
weakening of the body's immune system.
(3) Antibiotic therapy which can cause a wide range of microbial
changes. A microbe commonly residing in the intestine without harmful
effects may undergo a wide range of changes as a result of antibiotic
(4) Having taken oral contraceptives (these are comprised of steroid
(5) The use of cortiosteroids including hydrocortisone and
Prednisone and cortiosteroid enemas.
(6) A bad case of food poisoning or drinking contaminated water as
the result of traveling to foreign countries.
Once the normal equilibrium of the colon is disturbed for any
reason, its microbes can migrate into the small intestine and stomach
hampering digestion, competing for nutrients, and overloading the
intestinal tract with their waste products.
There has been a long history indicating that bacteria and yeast are
involved in intestinal disease.
Early researchers working on ulcerative colitis believed this
disorder to be caused by bacteria. From 1906 to 1924, numerous researchers
isolated certain types of bacteria, injected either the bacteria or the
bacterial toxins into laboratory animals, and claimed that the injections
produced ulcerative colitis in the animals. In 1932, when Dr. B.B. Crohn
spoke about a "new" intestinal disorder which he called regional
ileitis (now known as Crohn's disease), some physicians attending his
lecture stated that this new disease entity might be due to
From the 1920's until the present, the role of microbes and the
products they produce continues to be investigated in an effort to find
the cause of various forms of inflammatory bowel disease. Often there have
been very convincing evidence that particular bacteria could initiate a
certain type of intestinal disease but, eventually, the work has been
dismissed because of insufficient proof. Some of the difficulties which
these investigators experienced in trying to pinpoint the
"culprit" microbes were undoubtedly due to the ever-changing
conditions of the microbial world of the intestine, to variability in the
strains of intestinal microbes, or to the lack of precise laboratory
techniques of identification.
In the 1980's an increasing number of reports have been published
stating that intestinal bacterial toxins appear to be injuring intestinal
cells and, as a result, causing a variety of diarrheal diseases. Some of
the bacteria producing these toxins have not, in the past, been considered
to be disease producing types. Although there is still insufficient
evidence to link a specific microbe to each of the chronic intestinal
disorders, it is generally agreed that intestinal microbes are not
This was expressed by Gary Gitnick, M.D. of the University of
California School of Medicine, who wrote in Drug Therapy, December
While they are having their acute attacks, cultures are routinely
made on stools of colitis patients. In variably, these cultures are
reported as showing "no pathogenic organisms." Only the normal
bacterial inhabitants of the bowel are seen. One valuable clue to the
solution of the problem is that, while ulcerative colitis resembles an
infectious disease, no "disease" germs are detectable in the
bowel movements of the victims. The idea presented itself that perhaps the
"infection is caused by the bacteria that normally inhabit the bowel,
the "normal flora"; that these bacteria could be fermenting
undigested sugars that had not been absorbed from the bowel by its owner.
Even though affected people fail to digest some sugars, many of the myriad
of kinds of bacteria can. They ferment the sugars, producing chemicals
that irritate the bowel and cause inflammation that looks exactly like the
infections caused by recognized disease germs!
The infection concept is reinforced by the knowledge that attacks of
colitis can be prevented or made less severe by taking a poorly absorbed
sulfa drug such as Azulfidine. This successful prophylactic measure again
suggests that infection is involved in IBD-even though no disease germs
can be cultured.
Infection must play an important role in inflammatory bowel disease,
but some other factor or factors are involved. Something must be
operating, possibly some toxic element common in many foods (a single food
irritant would surely have been discovered long ago).
THE VICIOUS CYCLE
The presence of undigested and unabsorbed carbohydrates within the
small intestine can encourage microbes from the colon to take up residence
in the small intestine and to continue to multiply. This, in turn, may
lead to the formation of products, in addition to gas, which injure the
small intestine. Examples are lactic, acetic, and other acids which are
short-chain organic acids resulting from the fermentative process.
Once bacteria multiply within the small intestine, a chain of events
develops into a vicious cycle characterized by an increase in the
production of gas, acids and other products of fermentation which
perpetuate the malabsorption problem and prolong the intestinal disorder.
Bacterial growth in the small intestine appears to destroy the
enzymes on the intestinal cell surface preventing carbohydrate digestion
and absorption and making carbohydrates available for further
fermentation. It is at this point that excessive mucus may be triggered as
a self-defense mechanism whereby the intestinal tract attempts to
"lubricate" itself against the mechanical and chemical injury
caused by the microbial toxins, acids, and the incompletely digested and
unabsorbed carbohydrates. This further impairs digestion which, in turn,
allows more fermentation and thus the cycle continues to perpetuate.
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